| Your personal background. |
|---|
Vitreous haemorrhage generally happens suddenly, and without any pain. Symptoms range from the sudden appearance of floaters or spots in your vision, to a sudden blurring of vision, and in extreme cases, sudden blindness.
Some people find that their vision tends to be bad in the morning, as the blood as settled to back of their eye during the night.
What causes vitreous hemorrhage?
There are lots of possible causes of vitreous hemorrhage, including systemic diseases such as:
• Sickle cell anemia
• Diabetes mellitus
Also, with aging the vitreous gel liquefies and separates from the retina, making a posterior vitreous detachment. Bleeding can sometimes be linked when this happens. Other causes of vitreous hemorrhage contain retinal tears, ocular trauma or detachment, retinal vein occlusion, other tumors, vascular abnormalities, and rarely wet macular degeneration.
Vitreous Hemorrhage Symptoms
The primary symptoms of vitreous hemorrhage are cloudy and floaters vision. Floaters we associate with bleeding, patients explain as spider webs, lines, or many dark dots. If the vitreous hemorrhage is extremely important, there could be a big loss of vision. Whenever there has been a sudden onset of visual loss or floaters, careful retinal check is necessary both to diagnose the underlying cause of the vitreous bleeding and to determine if you need any specific therapy.
Is a vitreous hemorrhage painful?
No. this is one of the reasons why ophthalmologists are largely concerned about rising awareness about the situation. In many cases, a vitreous hemorrhage can develop extremely fast and without pain that indicates a medical emergency.
An ophthalmologist or nearby emergency room should be contacted promptly if signs of vitreous hemorrhages, such as sudden cobwebs or floaters in a vision or a shadowy or red effect, happen.
Treatment
The attendance of a retinal detachment may be determined using ultrasonography if a sufficeint view of the posterior segment is not possible. Vitrectomy is performed immediately when a retinal detachment or break is identified. Provided the retina is linked, observation is on an outpatient basis. If the view to the posterior pole is blocked, restriction of activities and elevation of the head of the bed while sleeping may permit the blood to settle inferiorly and allow visualization of the superior retina where retinal breaks most generally happen. Retinal breaks are sealed with laser photocoagulation or cryyotherapy. Retinal breaks are sealed with laser photocoagulation or cryotherapy. If a retinal detachment has been ruled out, patients may return to general activities.
Once the retina can be visualized, treatment is aimed at the underlying etiology as soon as easy. If neovascularization from proliferative retinopathy is the cause, laser panretinal photocoagulation is done, if possible via the residual hemorrhage, to cause regression of neovascularization. A Krypton laser may help photocoagulation as it passes via photocaogulation as it passes via hemorrhage better than argon lasers. An indirect laser system may also permit energy delivery to the retina around a vitreous hemorrhage. Alternatively, in the interim, intravitreal anti-VEGF agents may induce regression of the neovascularization until laser photocoagulation is easy. |
| Your feedback on this profile |
|---|
| Recommend this profile for User of the Day: |
I like this profile |
| Alert administrators to an offensive profile: |
I do not like this profile |
|
| Account data |
View |
| Team |
None |
|
